Condyloma acuminata (Genital warts) HPV infection transmitted through sexual contact, contact parts of the small trauma can promote infection, three kinds of squamous epithelium (skin, mucosa, metaplasia) on HPV infection rates are sensitive to. Each type of clinical HPV and special damages related to, and the squamous epithelium of the skin or mucous membranes have their predilection sites. Contains a relatively large number of virus particles when the shedding of superficial cells or keratin debris into the epithelium susceptible to fracture when the infection may arise, it can be a result of direct contact or a rare auto-inoculation or contamination of the underwear, bath tub, bath towel, bedpan infection.
Human cases of infection the virus can be lurking in the basal keratinocytes, the epidermal cell layer in the replication, HPV penetrated the nucleus, causing the rapid cell division, accompanied by the propagation and spread virus particles to form a characteristic papilloma. Late gene expression of structural polypeptides, namely, the structural protein assemble particles, the virus mainly concentrated in the granular layer of the cell nucleus, in the granular layer of the epidermis appear koilocytotisis increase in histological normal epithelial cells also have HPV, the residual DNA after treatment can often lead to disease recurrence.
HPV warts on the skin caused by redundant, in the throat, anal, genital mucosa to form proliferative lesions, the virus type is a small DNA virus. The lesions occur in the majority of HPV infections are benign and can be self-limiting, but there are worse cases. Such as the perianal, genital mucosa to form a flat cell carcinoma were reported. There are rare genetic skin disease, wart-like epidermal dysplasia superfluous (EV) renewal of skin cancer, and so, in the cancer cells detected HPV.
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