Genital wart is one kind of sexually transmitted diseases, which is caused by HPV—Human Papilloma Virus. There are several transmission ways. The most common way is sexual transmission. And people can also get genital warts from non-sexual causes. For example, touch the lesion distribution or the secretion of the patients. And there are other ways of transmission, such as indirect contact. If you touch the clothes or the things of the patient’s, you have a risk of getting genital wart.

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Female genital warts can be as small as 1mm

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At this post we post some genital warts images on parts of human body, to find more genital warts images please read on our blog.

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Female genital warts can be as small as 1mm

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I must tell you how I could know about the disease called Genital warts. A friend of mine has had genital warts for more than 5 years, and he never found a product that really worked. He lost his confidence, lost his happiness, and almost lost his family, because his wife thought he has dirty relationship with other women. By the way, I stand behind him during his worst time. This is why I know about genital warts.

In fact, genital wart is one kind of sexually transmitted diseases, which is caused by HPV—Human Papilloma Virus. There are several transmission ways. The most common way is sexual transmission. And people can also get genital warts from non-sexual causes. For example, touch the lesion distribution or the secretion of the patients. And there are other ways of transmission, such as indirect contact. If you touch the clothes or the things of the patient’s, you have a risk of getting genital wart.

What’s more, iatrogenic infection is another way of transmission. People can get the disease during the examination or surgery.

So, you can know that arbitrarily dismissing the direct link between sexual promiscuity and the spread of sexually transmitted diseases would be foolhardy. There are so many ways to get this bad disease.

After you get genital warts, it is very painful. The lesion distribution would be very terrible. And it is irritating and painful at the same time. Male patients usually have painful or difficult urination, while the female patients’ vulvae and vagina could be red, painful and urticant Sao. All of these symptoms are suffering.

If you or your friend is unfortunate enough to get genital warts, don’t be worry about it too much. Modern medicine could be very effective and has a great chance to cure genital wart. You can get got surgical treatment to get rid of it. The small scale genital wart is appropriate to do this surgery. Cold-cure treatment is another effective way. It uses liquid nitrogen of -196℃ to help the wart shed off, so you can clear the wart off. Female patients are appropriate to take laser treatment of CO_2 Laser, especially good for the warts in vulvae.

There are many good treatments to treat the genital warts, but people rely on modern medicine too much. If you don’t try your best to improve the physique, the modern treatments will not work very well on you. You must do physical exercise, and change your bad habit. Try to wash your underwear everyday by soap with clean warm water. And do not wash scour your vagina, because vagina has the ability of cleaning itself, if you scour it, you can ruin the good and healthy condition in the vagina.

Try to choose to be in a good mood at the same time, and you will get both your confidence and health back!

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Boys and young men, however, don’t get the same free coverage under the law, even though the human papillomavirus (HPV) vaccine is also approved for the prevention of genital warts in males.

Why the difference? “Genital warts aren’t life threatening,” says Debbie Saslow, director of breast and gynecologic cancer for the American Cancer Society.

Merck, which manufactures one of the two FDA-approved HPV vaccines, is conducting research to see if the vaccine prevents genital cancers in men, says Saslow. In the meantime, though, the vaccine has already been shown to prevent cervical, vaginal and vulvar cancers in women. “It’s a matter of cost-effectiveness,” says Saslow.

The vaccine is pricey, requiring three shots over a six-month period, at about $130 each.

The human papillomavirus, which is transmitted through sexual contact, is extremely common, accounting for an estimated 500,000 cases of genital warts every year. “It’s the common cold of the genital tract,” says Saslow.

There are several treatments to eliminate genital warts. And, the body can rid of the virus, although it’s possible to get re-infected.

The health law requires that immunizations that are recommended by the Advisory Committee on Immunization Practices be provided without charge to patients in new health plans starting this fall. ACIP is a group of 15 experts appointed by the Secretary of Health and Human Services.

While the HPV vaccine is recommended for all girls and young women between 9 and 26, the committee made a “permissive” recommendation for boys and men in the same age range, says Lance Rodewald, director of the immunization services division for the Centers for Disease Control and Prevention. Basically, that means it’s OK to vaccinate males for genital warts, but it’s not essential.

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Patient-Applied:

Podofilox 0.5% solution or gel. Patients should apply podofilox solution with a cotton swab, or podofilox gel with a finger, to visible genital warts twice a day for three days, followed by 4 days of no therapy. This cycle may be repeated, as necessary, for up to four cycles. The total wart area treated should not exceed 10 cm2, and the total volume of podofilox should be limited to 0.5 mL per day. If possible, the health-care provider should apply the initial medicine to demonstrate the proper application technique and identify which warts should be treated. The safety of podofilox during pregnancy has not been established.
OR
Imiquimod 5% cream. Patients should apply imiquimod cream once daily at bedtime, three times a week for up to 16 weeks. The treatment area should be washed with soap and water 6-10 hours after the application. The safety of imiquimod during pregnancy has not been established.

Provider-Administered:

Cryotherapy with liquid nitrogen or cryoprobe. Repeat applications every 1-2 weeks.
OR
Podophyllin resin 10%-25% in a compound tincture of benzoin. A touch should be applied to each wart and allowed to air dry. The treatment may be repeated weekly, if necessary. To avoid the possibility of complications associated with systemic absorption and toxicity, two important guidelines should be followed: 1) application should be limited to <0.5 mL of podo-phyllin or an area of <10 cm2 of warts per session, and 2) no open lesions or wounds should exist in the area to which treatment is administered. Some specialists suggest that the preparation should be thoroughly washed off 1-4 hours after application to reduce local irritation. The safety of podophyllin during pregnancy has not been established.
OR
Trichloroacetic acid (TCA) or Bichloroacetic acid (BCA) 80%-90%. A small amount should be applied only to the warts and allowed to dry, at which time a white “frosting” develops. If an excess amount of acid is applied, the treated area should be powdered with talc, sodium bicarbonate (i.e., baking soda), or liquid soap preparations to get rid of unreacted acid. This treatment can be repeated weekly, if necessary.
OR
Surgical removal either by tangential scissor excision, tangential shave excision, curettage, or electrosurgery

Alternative Regimens

Intralesional interferon
OR
Laser surgery

Podofilox 0.5% solution or gel, an antimitotic drug that destroys warts, is relatively inexpensive, easy to use, safe, and self-applied by patients. The majority of patients experience mild-to-moderate pain or local irritation after treatment. Imiquimod is a topically active immune enhancer that stimulates production of interferon and other cytokines. Local inflammatory reactions are common with the use of imiquimod; these reactions include redness and irritation and are usually mild to moderate. Traditionally, follow-up visits are not required for patients using self-administered therapy. However, follow-up might be useful several weeks into therapy to determine the appropriateness of medication use and the response to treatment.

Cryotherapy destroys warts by thermal-induced cytolysis. Health-care providers must be trained on the proper use of this therapy because over- and undertreatment might lead to complications or low efficacy. Pain after application of the liquid nitrogen, followed by necrosis and sometimes blistering, is common. Local anesthesia (topical or injected) might facilitate therapy if warts are present in many areas or if the area of warts is large.

Podophyllin resin, which contains several compounds, including antimitotic podophyllin lignans, is another treatment option. The resin is most frequently compounded at 10%-25% in a tincture of benzoin. However, podophyllin resin preparations differ in the concentration of active components and contaminants. The shelf life and stability of podophyllin preparations are unknown. A thin layer of podophyllin resin must be applied to the warts and allowed to air dry before the treated area comes into contact with clothing; overapplication or failure to air dry can result in local irritation caused by spread of the compound to adjacent areas.

Both TCA and BCA are caustic agents that destroy warts by chemical coagulation of proteins. Although these preparations are widely used, they have not been investigated thoroughly. TCA solutions have a low viscosity comparable with that of water and can spread rapidly if applied excessively; therefore, they can damage adjacent tissues. Both TCA and BCA should be applied sparingly and allowed to dry before the patient sits or stands. If pain is intense, the acid can be neutralized with soap or sodium bicarbonate.

Surgical therapy has the good thing about usually eliminating warts at just one visit. However, such therapy requires substantial clinical training, additional equipment, and a longer office visit. After local anesthesia is applied, the visible genital warts can be physically destroyed by electrocautery, in which case no additional hemostasis is required. Care must be taken to control the depth of electrocautery to prevent scarring. Alternatively, the warts can be removed either by tangential excision with a pair of fine scissors or a scalpel or by curettage. Because the majority of warts are exophytic, this procedure can be accomplished with a resulting wound that only extends into the upper dermis. Hemostasis can be achieved with an electrocautery unit or a chemical styptic (e.g., an aluminum chloride solution). Suturing is neither required nor indicated in the majority of cases if surgical removal is performed properly. Surgical therapy is most beneficial for patients who have a large number or area of genital warts. Carbon dioxide laser and surgery might be useful in the management of extensive warts or intraurethral warts, particularly for those patients who have not responded to other treatments.

Interferons, both natural or recombinant, have been used for the treatment of genital warts. They have been administered systemically (i.e., subcutaneously at a distant site or IM) and intralesionally (i.e., injected into the warts). Systemic interferon is not effective. The efficacy and recurrence rates of intralesional interferon are comparable to other treatment modalities. Administration of intralesional interferon is associated with stinging, burning, and pain at the injection site. Interferon is probably effective because of its antiviral and/or immunostimulating effects. Interferon therapy is not recommended as a primary modality because of inconvenient routes of administration, frequent office visits, and the association between its use and a high frequency of systemic adverse effects.

Because of the shortcomings associated with all available treatments, some clinics employ combination therapy (i.e., the simultaneous use of two or more modalities on the same wart at the same time). No data support the use of more than one therapy at a time to improve efficacy of treatment, and some specialists believe that combining modalities might increase complications.
Recommended Regimens for Cervical Warts

For women who have exophytic cervical warts, high-grade SIL must be excluded before treatment is initiated. Management of exophytic cervical warts should include consultation with a specialist.

Recommended Regimens for Vaginal Warts

Cryotherapy with liquid nitrogen. The use of a cryo-probe in the vagina is not recommended because of the risk for vaginal perforation and fistula formation.
OR
TCA or BCA 80%-90% applied to warts. A small amount should be applied only to warts and allowed to dry, at which time a white “frosting” develops. If an excess amount of acid is applied, the treated area should be powdered with talc, sodium bicarbonate, or liquid soap preparations to remove unreacted acid. This treatment can be repeated weekly, if necessary.

Recommended Regimens for Urethral Meatus Warts

Cryotherapy with liquid nitrogen
OR
Podophyllin 10%-25% in compound tincture of benzoin. The treatment area must be dry before contact with normal mucosa. This treatment can be repeated weekly, if necessary. The safety of podophyllin during pregnancy has not been established.

Although data evaluating the use of podofilox and imiquimod for the treatment of distal meatal warts are limited, some specialists recommend their use in some patients.
Recommended Regimens for Anal Warts

Cryotherapy with liquid nitrogen
OR
TCA or BCA 80%-90% applied to warts. A small amount should be applied only to warts and allowed to dry, at which time a white “frosting” develops. If an excess amount of acid is applied, the treated area should be powdered with talc, sodium bicarbonate, or liquid soap preparations to remove unreacted acid. This treatment can be repeated weekly, if necessary.
OR
Surgical removal

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has a 200-fold increased risk for the development of cervical cancer while one infected with HPV 6 (a low risk HPV) has a 10-fold increased risk. Notably, HPV 6 is the causative agent of the most prevalent viral sexually transmitted disease, benign hyperproliferative genital warts. Over a million new cases of this disease are diagnosed annually and almost four billion dollars in US health care costs are spent annually. HPVs replicate in the differentiated compartment of the epithelium. The viruses must, therefore, have the ability to either abrogate growth arrest that occurs when the infected cells differentiate or induce differentiated cells to re-enter the cell cycle. While an examination of the high risk viruses has been emphasized, it is imperative that we understand the mechanism of pathogenicity of the low risk viruses, clinically significant pathogens in their own right. Therefore, our focus is to define the interactions between HPV 6 and a differentiating epithelium that dictate the outcome of infection. Because of the interaction of E7 with pivotal cell cycle control proteins, E7 is likely to play a critical role in altering the intracellular environment in such a way as to promote the virus life cycle. For this reason, we have chosen to concentrate on this gene product. In Specific Aim 1 of the proposal we will establish the role of E7 in the HPV 6 life cycle. We will use the intact HPV 6 genome, with a translation termination linker in the E7 gene, to determine the necessity of 6E7 for a) episomal maintenance in primary undifferentiated keratinocytes during the non-productive stage of the life cycle, and b) the HPV-mediated aberrant cellular DNA synthesis in the suprabasal cell layer of organotypic raft cultures during the productive stage of the life cycle. We will also use the raft culture to determine the sufficiency of 6E7 in altering epithelial cell growth. In Specific Aim 2 we will conduct a structure/function analysis of the 6E7 protein to identify regions of E7 pertinent to the virus life cycle. We will analyze the mutants in the context of the intact genome for their necessity in episomal maintenance in undifferentiated cells and for reprogramming suprabasal cells to enter S phase. In Specific Aim 3 we will determine the cellular targets of 6E7 relevant to the life cycle of the virus, with emphasis on the pRb family:E2F-mediated pathways. The effect of 6E7, in the context of the entire genome, on cellular gene expression during the non-productive and productive stages of the life cycle, will be determined. By conducting these experiments under physiological conditions that mimic a natural infection, we expect to make novel contributions on the activities of this important protein.

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